Synaptopathy underlying memory space deficits in Alzheimers disease (Advertisement) is increasingly regarded as instigated by toxic oligomers from the amyloid beta peptide (AOs). the theory that physiological safety against synaptotoxic AOs could be mediated by astrocyte-derived insulin/IGF1, but that protection itself is usually susceptible to AO accumulation. Intro Alzheimers disease (Advertisement) may be the most… Continue reading Synaptopathy underlying memory space deficits in Alzheimers disease (Advertisement) is increasingly