Ataxia telangiectasia mutated (ATM) mediates DNA harm response by controling irradiation-induced foci formation, cell routine checkpoint, and apoptosis. (EGFR), a membrane-bound receptor tyrosine kinase and well-documented oncogene, features in regulating the mitogen-activated proteins kinase, phospholipase C, sign transducer and activator of transcription (STAT), and phosphatidylinositol-3 kinase pathways in tumor cells11. Furthermore to its function in… Continue reading Ataxia telangiectasia mutated (ATM) mediates DNA harm response by controling irradiation-induced