Faulty lung septation and angiogenesis, quintessential features of neonatal chronic lung disease (CLD), typically result from lengthy exposure of developing lungs to mechanical ventilation (MV) and hyperoxia. Lapatinib (free base) manufacture [VEGF-A, VEGF receptor 1 (VEGF-R1), VEGF-R2 immunoblots], TGF activation [phosphorylated Smad2 (pSmad2) quantitative-IHC], and elastin production (tropoelastin immunoblots, quantitative image analysis of Hart’s stained… Continue reading Faulty lung septation and angiogenesis, quintessential features of neonatal chronic lung